SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence. (2021)
Abstract
No abstract provided
Bibliographic Information
Digital Object Identifier: http://dx.doi.org/10.1182/blood.2020010432
PubMed Identifier: 33988691
Publication URI: http://europepmc.org/abstract/MED/33988691
Type: Journal Article/Review
Volume: 138
Parent Publication: Blood
Issue: 11
ISSN: 0006-4971