The increase in BIK expression following ERK1/2 pathway inhibition is a consequence of G1 cell-cycle arrest and not a direct effect on BIK protein stability (2014)
Attributed to:
How does ERK1/2-dependent phosphorylation target BimEL to the proteasome?
funded by
BBSRC
Abstract
No abstract provided
Bibliographic Information
Digital Object Identifier: http://dx.doi.org/10.1042/bj20131346
PubMed Identifier: 24527759
Publication URI: http://europepmc.org/abstract/MED/24527759
Type: Journal Article/Review
Parent Publication: Biochemical Journal
Issue: 3
ISSN: 0264-6021