Plasmid-based gap-repair recombineered transgenes reveal a central role for introns in mutually exclusive alternative splicing in Down Syndrome Cell Adhesion Molecule exon 4. (2019)

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1093/nar/gky1254

PubMed Identifier: 30541104

Publication URI: http://europepmc.org/abstract/MED/30541104

Type: Journal Article/Review

Volume: 47

Parent Publication: Nucleic acids research

Issue: 3

ISSN: 0305-1048