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The increase in BIK expression following ERK1/2 pathway inhibition is a consequence of G1 cell-cycle arrest and not a direct effect on BIK protein stability. (2014)

First Author: Sale MJ

Attributed to: How does ERK1/2-dependent phosphorylation target BimEL to the proteasome? funded by BBSRC

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1042/bj20131346

PubMed Identifier: 24527759

Publication URI: http://europepmc.org/abstract/MED/24527759

Type: Journal Article/Review

Volume: 459

Parent Publication: The Biochemical journal

Issue: 3

ISSN: 0264-6021