📣 Help Shape the Future of UKRI's Gateway to Research (GtR)

We're improving UKRI's Gateway to Research and are seeking your input! If you would be interested in being interviewed about the improvements we're making and to have your say about how we can make GtR more user-friendly, impactful, and effective for the Research and Innovation community, please email gateway@ukri.org.

HPV16 and 18 genome amplification show different E4-dependence, with 16E4 enhancing E1 nuclear accumulation and replicative efficiency via its cell cycle arrest and kinase activation functions. (2017)

First Author: Egawa N

Attributed to: Molecular Biology of Human Papillomavirus Infection 2 funded by MRC

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1371/journal.ppat.1006282

PubMed Identifier: 28306742

Publication URI: http://europepmc.org/abstract/MED/28306742

Type: Journal Article/Review

Volume: 13

Parent Publication: PLoS pathogens

Issue: 3

ISSN: 1553-7366