Norovirus infection results in eIF2a independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation. (2020)
Attributed to:
Dissecting the mechanism of translational control during calicivirus infection
funded by
BBSRC
Abstract
No abstract provided
Bibliographic Information
Digital Object Identifier: http://dx.doi.org/10.1371/journal.ppat.1008250
PubMed Identifier: 31905230
Publication URI: http://europepmc.org/abstract/MED/31905230
Type: Journal Article/Review
Volume: 16
Parent Publication: PLoS pathogens
Issue: 1
ISSN: 1553-7366