Neuron type-specific increase in lamin B1 contributes to nuclear dysfunction in Huntington's disease. (2021)
Attributed to:
Genome stability established through epigenome plasticity during ageing and rejuvenation
funded by
UKRI
Abstract
No abstract provided
Bibliographic Information
Digital Object Identifier: http://dx.doi.org/10.15252/emmm.202012105
PubMed Identifier: 33369245
Publication URI: http://europepmc.org/abstract/MED/33369245
Type: Journal Article/Review
Volume: 13
Parent Publication: EMBO molecular medicine
Issue: 2
ISSN: 1757-4676