📣 Help Shape the Future of UKRI's Gateway to Research (GtR)

We're improving UKRI's Gateway to Research and are seeking your input! If you would be interested in being interviewed about the improvements we're making and to have your say about how we can make GtR more user-friendly, impactful, and effective for the Research and Innovation community, please email gateway@ukri.org.

Homozygous GDF2 nonsense mutations result in a loss of circulating BMP9 and BMP10 and are associated with either PAH or an "HHT-like" syndrome in children. (2021)

First Author: Hodgson J

Attributed to: Mechanisms underlying the development of pulmonary arterial hypertension funded by MRC

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1002/mgg3.1685

PubMed Identifier: 33834622

Publication URI: http://europepmc.org/abstract/MED/33834622

Type: Journal Article/Review

Volume: 9

Parent Publication: Molecular genetics & genomic medicine

Issue: 12

ISSN: 2324-9269