📣 Help Shape the Future of UKRI's Gateway to Research (GtR)

We're improving UKRI's Gateway to Research and are seeking your input! If you would be interested in being interviewed about the improvements we're making and to have your say about how we can make GtR more user-friendly, impactful, and effective for the Research and Innovation community, please email gateway@ukri.org.

SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence. (2021)

First Author: Gao J

Attributed to: Joint Wellcome and MRC Centre, ‘Wellcome Trust – Medical Research Council Cambridge Stem Cell Institute’ funded by MRC

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1182/blood.2020010432

PubMed Identifier: 33988691

Publication URI: http://europepmc.org/abstract/MED/33988691

Type: Journal Article/Review

Volume: 138

Parent Publication: Blood

Issue: 11

ISSN: 0006-4971