Loss of the batten disease protein CLN3 leads to mis-trafficking of M6PR and defective autophagic-lysosomal reformation. (2023)
Abstract
No abstract provided
Bibliographic Information
Digital Object Identifier: http://dx.doi.org/10.1038/s41467-023-39643-7
PubMed Identifier: 37400440
Publication URI: http://europepmc.org/abstract/MED/37400440
Type: Journal Article/Review
Volume: 14
Parent Publication: Nature communications
Issue: 1
ISSN: 2041-1723