Loss of the batten disease protein CLN3 leads to mis-trafficking of M6PR and defective autophagic-lysosomal reformation. (2023)

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1038/s41467-023-39643-7

PubMed Identifier: 37400440

Publication URI: http://europepmc.org/abstract/MED/37400440

Type: Journal Article/Review

Volume: 14

Parent Publication: Nature communications

Issue: 1

ISSN: 2041-1723