Patient consent forms from A novel FAM83G variant from palmoplantar keratoderma patient disrupts WNT signalling via loss of FAM83G-CK1a interaction. (2024)
Attributed to:
TGFß and Wnt signalling during development and human disease
funded by
MRC
Abstract
No abstract provided
Bibliographic Information
Digital Object Identifier: http://dx.doi.org/10.6084/m9.figshare.26245748.v1
Type: Other