📣 Help Shape the Future of UKRI's Gateway to Research (GtR)

We're improving UKRI's Gateway to Research and are seeking your input! If you would be interested in being interviewed about the improvements we're making and to have your say about how we can make GtR more user-friendly, impactful, and effective for the Research and Innovation community, please email gateway@ukri.org.

Nucleosomal asymmetry shapes histone mark binding and promotes poising at bivalent domains. (2025)

First Author: Bryan E

Attributed to: To study epigenetic mechanisms regulating lineage commitment and cell potency funded by BBSRC

Abstract

No abstract provided

Bibliographic Information

Digital Object Identifier: http://dx.doi.org/10.1016/j.molcel.2024.12.002

PubMed Identifier: 39731917

Publication URI: http://europepmc.org/abstract/MED/39731917

Type: Journal Article/Review

Volume: 85

Parent Publication: Molecular cell

Issue: 3

ISSN: 1097-2765